They compare Sars-Cov-2 antibody duration with SARS/Mers in this study

https://www.nature.com/articles/s41591-020-0965-6

The strength and duration of immunity after infection are key issues for ‘shield immunity’¹⁸ and for informing decisions on how and when to ease physical distancing restrictions^19,20. Previous studies have shown that circulating antibodies against SARS-CoV or MERS-CoV last for at least 1 year^21,22. Sustained IgG levels were maintained for more than 2 years after SARS-CoV infection^23,24. Antibody responses in individuals with laboratory-confirmed MERS-CoV infection lasted for at least 34 months after the outbreak²⁵.

Recently, several studies characterizing adaptive immune responses to SARS-CoV-2 infection have reported that most COVID-19 convalescent individuals have detectable neutralizing antibodies, which correlate with the numbers of virus-specific T cells^26,27,28,29. In this study, we observed that IgG levels and neutralizing antibodies in a high proportion of individuals who recovered from SARS-CoV-2 infection start to decrease within 2–3 months after infection. In another analysis of the dynamics of neutralizing antibody titers in eight convalescent patients with COVID-19, four patients showed decreased neutralizing antibodies approximately 6–7 weeks after illness onset³⁰.

One mathematical model also suggests a short duration of immunity after SARS-CoV-2 infection³¹. Together, these data might indicate the risks of using COVID-19 ‘immunity passports’ and support the prolongation of public health interventions, including social distancing, hygiene, isolation of high-risk groups and widespread testing. Additional longitudinal serological studies profiling more symptomatic and asymptomatic individuals are urgently needed to determine the duration of antibody-mediated immunity. In addition, low levels of anti-viral IgG in asymptomatic patients, who might be more likely to become seronegative, further support the need for timely serosurvey to study the true infection rate.

Preliminary studies suggest that immunity from SARS-COV-2 is shorter and less effective than for SARS and MERS. It's still early days though, it has only been 3 months since the patients in this study got infected. Asymptomatic patients lose the immunity IgG anti bodies a lot faster and I imagine they also produced fewer T-cells against Sars-Cov-2.



This is also a problem for the older population
https://www.ctvnews.ca/health/coronavirus/covid-19-vaccine-may-not-work-in-older-people-experts-say-1.4997928

A vaccine against COVID-19 may not be as effective in older people who are most at risk of suffering complications and dying from the disease, according to U.K. researchers.

Dr. Eleanor Fish, an immunology professor at the University of Toronto, told CTVNews.ca that a coronavirus vaccine may not work on the elderly because their immune systems are "not as robust" as those of younger people. "If you are immunocompromised or if there is any way your immune system is weaker than it should be, then your body might not have the ability to adequately respond to the vaccine," Fish explained in a telephone interview on Wednesday.

When considering the seasonal flu shot, Fish said the elderly are typically given a larger dose of the flu vaccine so their immune systems have a better chance of recognizing and responding to the vaccine.

However, she warns that there are still a lot of unknowns in regards to a COVID-19 vaccine. "We don't know that if any of the vaccines being developed are going to be effective. We don't know whether... the key antibodies and the neutralizing antibodies are going to be effective against the virus," Fish said "But if we assume that there is going to be an effective vaccine [for] healthy adults and presume that healthy adults take the vaccine, there is still the potential that the vaccine my not prompt a response in the elderly."

The solution, immunize the younger population first to protect the elderly.



The question, will people co-operate?

This morning I went to the chiropractor again, more people there today, zero masks. I had mine on, cleaning my hands before and after touching things. Most people just clean after. Then to the supermarket, at least nearly half the shoppers wore masks there, but with a clear divide. The older (50+) people were all using masks, none of the younger people, none of the staff. An elderly couple thanked me for patiently waiting at a distance for them to make a selection and/or for wearing a mask. The staff was just working close together stocking shelves, no gloves to handle things. They did have sanitizer at the check out and plastic shields up to separate them from the customers. There were also distance markers and arrows on the ground and one person counting people going in and out (no line up this morning). Get a cart on one side, put it away on the other side for cleaning. (I cleaned it myself anyway)

If you're over 50 you're between a rock and a hard place. A vaccine might not be effective or can potentially make you sick anyway. Young people care more about their conveniences than your well being. You still got to eat, can't avoid exposure by locking yourself away.

The good news from Luxembourg:

- It's been a month since anybody died from the virus in the country.
- Mass testing is well on the way now. I got tested 2 weeks ago and my wife will get tested tomorrow. In the last 3 weeks we tested more people than in the whole 3 months before.
- Nobody is in intensive care anymore.

The bad news:

- Active cases keep rising again, and probably not just due to coronavirus. Right now we're on 73, up from just 28 two weeks ago.
- The number of positive tests per day keeps slowly rising, too, (but thankfully not at any alarming rate - yet).

https://msan.gouvernement.lu/en/graphiques-evolution.html#sg

A Palm Beach County Board of County Commissioners public comment session went viral Wednesday after residents denounced mandatory masking laws as “devil’s laws” that would “throw God’s wonderful breathing system out the door.”

Sylvia Ball (florida resident) - “very sad to see the authorities stomping on our constitutional rights,” adding, “They want to throw God's wonderful breathing system out the door. You’re all turning your backs on it,”

https://www.washingtonpost.com/us-policy/2020/06/25/irs-stimulus-checks-dead-people-gao/

Berlin is fine. It's a big city. There was one house in Neukölln which is under quarantine now. 

No sane person goes to Neukölln anyway.

MERS is often held by medical studies as having an infamously poor antibody response that isn't always readily detectable. To a lesser extent, that was also the case with SARS:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5512479/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4880093/

The research that the article mentions does refer to detection after 34 months, albeit at reduced levels and taking only select cases (a grand total of seven people) that had detectable antibodies to begin with, to develop a follow-up study:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5038413/

Why they have decided to present it as some sort of consensus or ordinary feature of MERS? No idea. You are free to draw your own conclusions.

Mind you, the same difficulties concerning neutralizing antibody titers and coronaviruses have been noticed all the way back to the 70s:

https://www.cambridge.org/core/services/aop-cambridge-core/content/view/77BBFFC8CDBB77DAEDB4BEBCA3D0F14F/S0022172400022294a.pdf/coronavirus_antibody_titres_in_sera_of_healthy_adults_and_experimentally_infected_volunteers.pdf

Of course, with MERS and others, the negative results were often credited to issues with the methods used instead of automatically being credited to a proper response being inexplicably absent, since empirical evidence showcases even a mild coronavirus disease protect you for at least about a year of the same virus. One would think that is also the case here, considering how novel the disease and all the tools we have are and how much, time and time again, false negatives have been shown to abound relative to SARS-CoV-2 testing.

I understand people want to be careful around this, but some people are acting as if it is the first time a pandemic has happened and nobody knows if the world will end or not.

Edit - I haven't found the paper itself, but I've seen a news article where an immunologist mentions a small study on recently infected people found most of them had memory cells but no (detectable) antibodies. So one wouldn't necessarily follow the other or depend on its prevalence too.

Trump inherited more ventilators (16,660) than have been distributed by the administration in this pandemic, despite repeatedly lying that he was left with nothing by Obama. Shocker.

New Jersey just added 1,854 probable COVID-19 deaths.

https://whyy.org/articles/n-j-coronavirus-recovery-new-unemployment-claims-tick-up/

Just watching a video of john campbell were he says that, one of the "Long term" side effects of getting covid19 is supposedly Diabetes.
Its only anecdotal so far, ei. people have noticed what appears to be a uptrend in diabetes, amoung people that have had covid19 and recovered.
(this doesnt mean everyone that gets covid, gets diabetes, just the chances of you developeing it, rise afterwards, like it can trigger it)

Apparently they also saw this same trend when they did experiments with infecting mice, with covid19.
Time to invest in a drug company that makes insulin I guess.

Also pulmonary fibrosis (lunge scarring).
Which is apparently common, even if you barely have symptoms, and recover, afterwards your lunges arnt as great as they were before you got covid19.

I'm more afraid of the opposite, people not taking it seriously enough and assuming herd immunity will solve the pandemic or it will just go away like sars and mers. The world won't end, but as my wife said (sarcastically), governments just want to let it simmer and kill off some of the elderly to relieve the pension funds. The current focus around the world (except New Zealand and Australia) is to get the economy back up asap, not waiting until the hard to detect asymptomatic cases are stomped out. Just look at the Netherlands now jumping straight to phase 3 while Germany just had a big flare up. Getting people back into planes is more important than protecting those that build our society.

Agreed, better, longer, bigger studies are needed, and more time is needed as well. One thing we have now (unfortunately) is the largest test bed in modern history to study a pandemic. Atm the possibility of re-infection / how well immunity protects you from getting infected (again) is simply unknown. The only 'evidence' atm is that monkeys infected with sars-cov-2 after receiving the Astrazeneca vaccine all got infected anyway. (But received a much larger dose than you would normally get exposed to, and it still prevented pnuemonia at least) https://www.independent.co.uk/news/health/coronavirus-vaccine-oxford-trials-concerns-transmission-astrazeneca-latest-a9521241.html

No chance it's from eating far less healthy during quarantine?
The causes of diabetes are pretty similar to how a lot of people are spending their time during the isolation :/

• High-fat and carbohydrate diet.
• High alcohol intake.
• Sedentary lifestyle.
• Obesity or being overweight.

Ok, it's not just anecdotal

https://www.sciencedaily.com/releases/2020/06/200612172220.htm

Clinical observations so far show a bi-directional relationship between COVID-19 and diabetes. On the one hand, diabetes is associated with increased risk of COVID-19 severity and mortality. Between 20 and 30% of patients who died with COVID-19 have been reported to have diabetes. On the other hand, new-onset diabetes and atypical metabolic complications of pre-existing diabetes, including life-threatening ones, have been observed in people with COVID-19.

It is still unclear how SARS-Cov-2, the virus that causes COVID-19, impacts diabetes. Previous research has shown that ACE-2, the protein that binds to SARS-Cov-2 allowing the virus to enter human cells, is not only located in the lungs but also in organs and tissues involved in glucose metabolism such as the pancreas, the small intestine, the fat tissue, the liver and the kidney. Researchers hypothesise that by entering these tissues, the virus may cause multiple and complex dysfunctions of glucose metabolism. It has also been known for many years that virus infections can precipitate type 1 diabetes.